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The arterioles and capillaries of the body become obstructed by the resulting complexes of activated platelets , which have adhered to the endothelium via large multimeric vWF. Through a mechanism known as microangiopathic hemolysis , the growing thrombi lodged in smaller vessels destroy red blood cells RBCs as they squeeze through the narrowed blood vessels, forming schistocytes , or fragments of sheared RBCs. Shiga-toxin directly activates the alternative complement pathway and also interferes with complement regulation by binding to complement factor H, an inhibitor of the complement cascade.
Shiga-toxin causes complement-mediated platelet, leukocyte, and endothelial cell activation, resulting in systemic hemolysis, inflammation and thrombosis. However, in comparison to TTP, the kidneys tend to be more severely affected in HUS, and the central nervous system is less commonly affected.
Histologically , the glomeruli show thickened and sometimes split capillary walls due largely to endothelial swelling. You can read the full text of this article if you: Your Name: Your Email:. Colleague's Email: Separate multiple e-mails with a ;. Send a copy to your email.
Some error has occurred while processing your request. Please try after some time. Coleman, Joshua F. January - Volume 34 - Issue 1 - p The American Journal of Surgical Pathology34 1: Add Item s to: An Existing Folder. A New Folder. The item s has been successfully added to " ".